Low-GPX4 drives a sustained drug-tolerant persister state in TNBC by a targetable adaptive FSP1 upregulation.

Overlay images of the red and green channels from immunofluorescence co-staining demonstrated distinct patterns of expression, where analysis of MOC K1 and K2 indicated a highly significant negative correlation (Spearman's correlation, −0.8767, p < 0.0001) between GPX4 and Vimentin protein levels ( Fig. 5 K).

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