Furthermore, their association with highly significant KEGG pathways, particularly ECM-receptor interaction and complement-coagulation cascades, might correlate these glycoprotein alterations to the immune response, tissue integrity, and inflammatory processes central to UC pathogenesis.
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Quantitative proteomic and glycoproteomic analysis identifies CLCA1, FBN1, and FGB as potential biomarkers for ulcerative colitis.
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