Endothelial-Derived CCL7 Promotes Macrophage Polarization and Aggravates Septic Acute Lung Injury via CCR1-Mediated STAT1 Succinylation.

Single‐cell sequencing data further confirmed that CCL7 was predominantly expressed in pan‐vascular endothelial cell clusters (Figure 1E ), and pseudo‐time analysis demonstrated an increasing trend of CCL7 expression in mouse pan‐vascular endothelial cells during sepsis (Figure S2C , Supporting Information).

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