Decoding the hypoxia-exosome-immune triad in OSA: PRCP/UCHL1/BTG2-driven metabolic dysregulation revealed by interpretable machine learning.

Front Immunol · 2025 · PMC12597927 · PMID 41221287

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These findings were consistently reproduced in independent experimental sets ( Figures 7D–F , SW872 human adipocytes; Figures 7G–I , eWAT), where PRCP upregulation was highly significant ( Figures 7D, G ), UCHL1 expression was robustly increased ( Figures 7E, H ), and BTG2 levels were consistently downregulated across all comparisons ( Figures 7F, I ).

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