The activities of the mitochondrial respiratory chain complex presented an increasing trend in the OE-NAT10 group and a decreasing trend in the sh-AdipoR1 + OE-NAT10 group after Ang II treatment (Fig. 7 K).
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NAT10 induces N4-acetylcytidine modification of AdipoR1-mediated mitochondrial biogenesis against endothelial-to-mesenchymal transition in hypertension.
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