Lack of intestinal Mucin-1 impairs intestinal epithelial barrier and promotes metabolic dysfunction-associated steatotic liver disease in male mice.

Compared with the overexpression of wild-type MUC1 in intestinal epithelia, the overexpression of MUC1ΔCT did not lead to an extremely significant increase in β-Catenin levels, but only maintained the physiological level of β-Catenin (Supplementary Fig. 5e ).

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