Integrating causal human genetics and <i>In vivo</i> transcriptomics to uncover a shared lipid-centric architecture in metabolic and neurocognitive disease.

While this dementia-to-NAFLD protective link represents a highly significant finding, its unidirectional nature places it in a distinct mechanistic category from the reciprocally-linked metabolic-neurocognitive axes that form the core of our investigation.

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