Interestingly, while the overall levels of circulating LPCs exhibited a decreasing trend in our diabetic model, with both saturated and polyunsaturated species, the level of LPC 20:4, carrying pro-inflammatory arachidonic acid, was significantly elevated in the diabetic group.
← all excerpts
Lysolecithin reprogramming via LPCAT1 modulation restores endothelial function and prevents diabetes-associated dysmetabolism.
1
—
—