Chronic-phase clustering was driven by significant increased MMP-3, MCP-3 and MCP-2, with an increasing trend in MIP-1α, MCP-5 (CCL12), and IL-16 mediators, which are associated with extracellular matrix turnover and chronic inflammatory signaling.
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Senescence-Driven Inflammation and Immune Dynamics in the Progression of Radiation Cystitis.
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