Further comparison revealed that, although the content of collagen I in the Apo-EVs treated group also increased, the increase was not as significant as that in the VEGF treated group.
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Apoptotic vesicles from endothelial cells promote endothelial progenitor cell differentiation and angiogenesis via miR-30a-5p mediated activation of the EGFR/PI3K/AKT/VEGF pathway.
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