Met32 governs transcriptional control of sulfur metabolic flexibility and resistance to reactive sulfur species in the human fungal pathogen <i>Candida albicans</i>.

mBio · 2026 · PMC13170222 · PMID 42003610

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highly significantno p-value reported

Given the importance of sulfur in biological systems, the ability of fungal pathogens to uptake and utilize both organic and inorganic sulfur-containing molecules may be highly significant for their fitness.

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Met32 governs transcriptional control of sulfur metabolic flexibility and resistance to reactive sulfur species in the human fungal pathogen &lt;i&gt;Candida albicans&lt;/i&gt;.

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Met32 governs transcriptional control of sulfur metabolic flexibility and resistance to reactive sulfur species in the human fungal pathogen <i>Candida albicans</i>.