Inflammatory bowel disease-induced inflammation augments clonal hematopoiesis of indeterminate potential through Ref-1.

For the MR analysis of the opposite direction (IBD to CHIP), we observed a close to null effect (OR IVW , 0.98; 95% CI, 0.96-1.00) of CD to CHIP with borderline significance ( P = .03; supplemental Table 12 ), compared with the CHIP-to-IBD direction (OR IVW , 1.15; 95% CI, 1.03-1.27), suggesting a potential bidirectional relationship.

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At the third DSS cycle, monocyte numbers showed an increasing trend in D3a -mutant mice compared with WT mice after DSS treatment, which were significantly elevated relative to vehicle-treated D3a -mutant mice ( Figure 7 B).

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