Differential Activation of Pro-Survival Pathways by NIX/BNIP3L: An Expression-Level-Dependent Mechanism Governing PC12 Cell Fate During H<sub>2</sub>O<sub>2</sub>-Induced Oxidative Stress.

Biology (Basel) · 2026 · PMC13255857 · PMID 42274518

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an increasing trendno p-value reported

ARMCX3, as a mitochondrial outer membrane protein, showed an increasing trend (in NIX-knockdown cells) consistent with TOMM20 and others, but the distinct behavior of VDAC may be related to its specific localization and function at mitochondria-associated endoplasmic reticulum membranes (MAMs).

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Differential Activation of Pro-Survival Pathways by NIX/BNIP3L: An Expression-Level-Dependent Mechanism Governing PC12 Cell Fate During H&lt;sub&gt;2&lt;/sub&gt;O&lt;sub&gt;2&lt;/sub&gt;-Induced Oxidative Stress.

The sentences

Differential Activation of Pro-Survival Pathways by NIX/BNIP3L: An Expression-Level-Dependent Mechanism Governing PC12 Cell Fate During H<sub>2</sub>O<sub>2</sub>-Induced Oxidative Stress.