Systemic TGF-β1 reduction contributes to neuronal GLUT4 trafficking impairment in acute hepatic encephalopathy

This cytosolic accumulation was not accompanied by a corresponding protein increase in the membrane fraction; instead, a decreasing trend in membrane-associated GLUT4 content was noted in the sTGF-β1n model ( Figure 3D ).

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We also documented a pronounced trend toward an increase in the neuronal glucose transporter GLUT3, which stabilizes neuronal glucose uptake and utilization, in cerebral cortex homogenates from AOM mice ( Figure 3C ).

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