Nuclear Lamina Dysfunction and DNA Damage as Drivers of Premature Senescence in a Human Müller Glial Cell Model of Spinocerebellar Ataxia Type 7.

A decreasing trend in RTL that was not statistically significant was determined in 64Q cells at seven days of dox-mediated induction of polyQ ataxin-7 ( Figure 3 e).

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An increasing trend in p21 mRNA expression levels, which lacked statistical significance, was observed in 64Q cells ( Figure 4 b) in comparison to 10Q cells.

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