Notwithstanding these limitations, our study’s key finding that hepatocyte-derived S100A4/A6/A10/A11 proteins serve as critical nodes connecting inflammation and fibrosis within the hepatic fibrotic network remains highly significant.
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Hepatocyte-Derived S100A4/A6/A10/A11 Proteins Promote Liver Fibrosis by Direct and Indirect Activation of HSCs.
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