Engineered exosomes improve myocardial cell membrane integrity and heart function in dystrophic cardiomyopathy.

Although the left ventricular wall thickness of the DmdΔ4 mice showed a decreasing trend at 40 weeks of age, the left ventricular volume remained basically unchanged as compared to the control mice (Figures 1N–P and S2H ).

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Moreover, the Exo‐CHP treatment also significantly increased the cardiac sarcospan protein levels in mice, but integrin β, although showing an increasing trend, did not show a statistically significant difference (Figure S14 ).

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