Molecular response to aromatase inhibitor treatment in primary breast cancer.

Genes in the 'proliferation cluster' exhibited a highly significant overlap with a previously characterized breast cancer proliferation signature [ 16 ].

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There was only a modestly significant relationship between the GIDE and the pretreatment level of ER based on immunohistochemistry; the current data suggest that high ER expression may be necessary for a tumour to be highly responsive (high GIDE) but that some tumours with a high ER have only a moderate or poor biological response.

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