TUTTLE 583 ance, like suppression of antibacterial resistance, is caused by a factor in circulation will appear in a forthcoming publication. In spite of the acquisition of a mechanism of macrophage-mediated resistance to a tumor cell challenge, the host remained unable to reject its primary tumor. It is highly significant in this connection that the factor which interferes with macrophage function continues to be produced and liberated into the circulation during growth of the primary tumor.
← all excerpts
Subversion of host defense mechanisms by murine tumors. II. Counter-influence of concomitant antitumor immunity.
1
—
—