The activating KIR genes, KIR2DS1 and KIR2DS5 , showed a weak association with increased risk of CIN3/cancer that bordered on significance (Table S2), possibly because of their strong linkage disequilibrium with KIR3DS1 ( 15 , 16 ).
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Hierarchy of resistance to cervical neoplasia mediated by combinations of killer immunoglobulin-like receptor and human leukocyte antigen loci.
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