Distinct cell-specific control of autoimmunity and infection by FcgammaRIIb.

5, C, D, and F ), whereas there was a nonsignificant trend toward increased histological joint damage, perhaps because of a failure of macrophage remodeling and/or immune complex clearance ( Fig. 5 H ).

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FcγRIIb overexpression on macrophages does not influence the T-dependent immune response Macrophage-transgenic (M-TG) mice were immunized with two T-independent antigens (NP-dextran and NP-LPS), and although a possible trend toward a reduction in IgG production was seen at later time points, initial responses to these antigens were not significantly different from control mice (Fig.

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