The interaction of vasoactive substances during exercise modulates platelet aggregation in hypertension and coronary artery disease.

There was no significant difference between the three groups at rest but at the peak of exercise patients with CAD had lower 6-keto-prostaglandin-F1a/thromboxane B 2 ratio compared with healthy volunteers that almost reached statistical significance (p = 0.053) (Figure 3C ).

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