GFP-MK/LisHAA and GFP-MKT723A had similar highly significant activities to GFP-MK in restoring normal cell morphology, whereas GFP-MKT723D and GFP-MKΔC35 were inactive ( Fig. 8 E ).
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Novel role of the muskelin-RanBP9 complex as a nucleocytoplasmic mediator of cell morphology regulation.
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