A novel transgenic mouse model of CBS-deficient homocystinuria does not incur hepatic steatosis or fibrosis and exhibits a hypercoagulative phenotype that is ameliorated by betaine treatment.

Mol Genet Metab · 2010 · PMC2954364 · PMID 20638879

Europe PMC DOI

hedged sentence

0.0010

closest p · 0.0× alpha

0.0010

boldest claim

The sentences

highly significantP = 0.001actually significant

4 B) that the HO mice exhibit a highly significant improvement in terms of survival and longevity compared to the MKO cbs (−/−) mice ( P = 0.001).

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