There was a slight trend toward lowered Aβ40/42 levels in FA-treated brain homogenates, and raised Aβ40/42 levels in DEA-treated brain homogenates for 6D11 treated Tg animals; however, the differences were not statistically significant.
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Anti-PrPC monoclonal antibody infusion as a novel treatment for cognitive deficits in an Alzheimer's disease model mouse.
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