STOX1: Key player in trophoblast dysfunction underlying early onset preeclampsia with growth retardation.

But, when looking at recurrent preeclampsia patients, a trend towards significance was detected in H-allele transmission when also corrected for the potential of a parent-of-origin effect [ 7 ].

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The correlation between these studies was found to be highly significant looking at genes affected by STOX1- and preeclampsia-modified genes [ 5 ].

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