Rett syndrome: genes, synapses, circuits, and therapeutics.

Front Psychiatry · 2012 · PMC3346964 · PMID 22586411

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an interesting trendno p-value reported

The molecular composition of synaptic NMDA receptor subunits showed an interesting trend in these MeCP2 KO animals: GluN2B-to-GluN2A subunit switching, which regulates the channel kinetics and biophysical properties of excitatory synapses in the developing brain, shows a delayed postnatal maturation and may be responsible for the molecular pathology of synaptic defects in RTT (Asaka et al., 2006 ).

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