Turner syndrome and the evolution of human sexual dimorphism.

However, this marginally nonsignificant difference was due to a significantly higher incidence of the nonmosaic 46,XiXq karyotype in Xpat females; when this karyotype category is excluded, the frequency of mosaics is essentially the same in both Xmat (15%) and Xpat (18%) females for their sample.

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Instead, the height of Turner syndrome females shows a strong, positive, highly significant correlation with their mother’s height (regardless of parental origin of the X), but the association with father’s height is smaller or nonexistent ( Salerno and Job 1987 ; Chu et al. 1994 ; Tsezou et al. 1999 ; Hamelin et al. 2006 ; Bondy et al. 2007 ; Kochi et al. 2007 ).

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