Epstein-Barr virus latent membrane protein 2A exacerbates experimental autoimmune encephalomyelitis and enhances antigen presentation function.

The contribution of latently infected B cells as antigen presenting cells may be significant in the context of MS, as myelin antigen would be limiting during the initiation of disease and B cells have been shown to be important antigen presenting cells when antigen is limiting 31 32 .

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