Twist1 suppresses senescence programs and thereby accelerates and maintains mutant Kras-induced lung tumorigenesis.

Notably, after performing gene set enrichment analysis (GSEA) [36] with this dataset, we found CT ON lungs had a global gene expression pattern that had a highly significant similarity to two overlapping gene sets for EMT [37] ( Figure 1D and Figure S1A ) and three EMT related phenotypes (hypoxia, metastasis and wound healing [38] , [39] , [40] ; Figure S1B – S1D ).

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