Focusing on the models for the estrogen-positive and estrogen-negative subsamples the essential finding for the Uppsala cohort is that proliferation and stromal-decorin are independently prognostic for estrogen-positive tumors, but not for estrogen-negative ones, whereas stromal-laminin is either significant or marginally significant for the estrogen-negative subpopulation.
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Stromal genes add prognostic information to proliferation and histoclinical markers: a basis for the next generation of breast cancer gene signatures.
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