Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding.

Leukemia · 2012 · PMC3419980 · PMID 22343733

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highly significantno p-value reported

Most importantly, in contrast to t(8;21) cells, the RUNX1 peaks unique for KN-AML showed a highly significant enrichment for binding sites for inducible factors such as AP1, C/EBP and NF-κB ( Figures 3d and e ).

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