Rac-induced left ventricular dilation in thyroxin-treated ZmRacD transgenic mice: role of cardiomyocyte apoptosis and myocardial fibrosis.

Furthermore, T4-stimulated myocardial Rac expression in transgenic mice was associated with highly significant increases in myocardial Rac-GTP activity and O2· − levels (14.10±2.24 and 3.70±0.15; p<0.001), respectively, compared to all other groups ( Figure 1B, C, D, E ).

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