HCMV-infected cells maintain efficient nucleotide excision repair of the viral genome while abrogating repair of the host genome.

After correcting for multiple statistical tests on the same data, the difference between 0 and 24 hp irradiation in the signal intensity within the host DNA was not significant (F = 3.1; df = 1, 123; p-value>0.16), whereas the difference between 0 and 24 hp irradiation in the signal intensity in the virus DNA was highly significant (F = 64.5; df = 1, 109; p-value<0.0002).

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