Genome-wide association studies have demonstrated highly significant associations between polymorphisms in IL-23 pathway genes, notably IL-23R, to a number of chronic inflammatory disorders, including inflammatory bowel disease (IBD) [2] , psoriasis [3] , and ankylosing spondylitis [4] .
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Age and CD161 expression contribute to inter-individual variation in interleukin-23 response in CD8+ memory human T cells.
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