TNNI3K, a cardiac-specific kinase, promotes physiological cardiac hypertrophy in transgenic mice.

PLoS One · 2013 · PMC3589374 · PMID 23472207

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highly significantno p-value reported

Since the phosphorylation of troponin is a highly significant element in the control of cardiac contractility, these data suggested the role of TNNI3K in regulating cTnI phosphorylation and contractile function in the heart.

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