N-Methyl-d-Aspartate Receptor - Nitric Oxide Synthase Pathway in the Cortex of Nogo-A-Deficient Rats in Relation to Brain Laterality and Schizophrenia.

Although we did not find similar significant changes in old genetically modified animals, some results displayed borderline significance here (e.g., correlations between NR1 in the R side and iNOS laterality, between NR1 laterality and iNOS in the R side and between NR1 laterality and iNOS laterality, see Table 4 ).

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Since these alterations in the cortical NMDAR–NOS pathway of young Nogo-A-deficient rats are very similar to those of old controls (despite the changes being only borderline significant in old controls), the data could be interpreted as accelerated aging of the young genetically modified animals.

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