Is there a role for treating inflammation in moyamoya disease?: a review of histopathology, genetics, and signaling cascades.

In particular, they failed to demonstrate the previous association of rs1800471 and tendency toward significance of rs1800470 suggesting that although TGFβ1 may be aberrantly expressed at the protein level this may be a result of stimulation from upstream mediators rather than mutations within the gene itself ( 48 ).

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