Modulation of vasodilator response via the nitric oxide pathway after acute methyl mercury chloride exposure in rats.

Biomed Res Int · 2013 · PMC3760274 · PMID 24024199

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failed to attain significanceno p-value reported

Though an increase in NO was observed in our previous study, it failed to attain significance as reductions in NO bioavailability may be due to the increased ROS production, which was responsible for the endothelial dysfunction [ 10 , 11 ].

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