Intragenic DNA methylation modulates alternative splicing by recruiting MeCP2 to promote exon recognition.

Cell Res · 2013 · PMC3817542 · PMID 23938295

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We further show that inhibition of histone deacetylase (HDAC) activity leads to exon skipping that shows a highly significant degree of overlap with that caused by MeCP2 knockdown.

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