recently described a STAT5A and ABL-tyrosine kinase dependent increase in ROS production and a highly significant and clinically relevant correlation between STAT5A expression and mutation status of BCR-ABL [37] .
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recently described a STAT5A and ABL-tyrosine kinase dependent increase in ROS production and a highly significant and clinically relevant correlation between STAT5A expression and mutation status of BCR-ABL [37] .