Slow repair of lipid peroxidation-induced DNA damage at p53 mutation hotspots in human cells caused by low turnover of a DNA glycosylase.

Nucleic Acids Res · 2014 · PMC4132702 · PMID 25081213

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highly significantno p-value reported

As these referred studies highlight the significance of BER glycosylase activity in cancer prevention, it is highly significant that we observed sequence-specific excision deficiency in a normal, wild-type glycosylase.

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