Accordingly, we found a highly significant difference (in early stages) in Bmf expression and a small not-significant difference in levels of Bcl-2 , Bcl-xl , and Bim , suggesting that while Bim would be only relevant in CPCs, Bmf might mediate the protective role of miR-133 against stress-induced apoptosis, both in CPCs and cardiomyocytes.
← all excerpts
miR-133a enhances the protective capacity of cardiac progenitors cells after myocardial infarction.
1
—
—