Although we observed a slight trend towards increased numbers of BrdU + O4 + cells in the SGZ of anti-EGF Ab-treated EAE mice, this trend did not reach a level of significance (0.6 ± 0.1 in the anti-EGF Ab-treated group versus 0.4% ± 0.02 in the IC-treated group, P = NS, Figures 4(a) , 4(b) , and 4(c) ).
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Treatment with Anti-EGF Ab Ameliorates Experimental Autoimmune Encephalomyelitis via Induction of Neurogenesis and Oligodendrogenesis.
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