Redox regulation of cardiomyocyte cell cycling via an ERK1/2 and c-Myc-dependent activation of cyclin D2 transcription.

J Mol Cell Cardiol · 2015 · PMC4312357 · PMID 25450615

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highly significantno p-value reported

It may also be highly significant that inappropriate ERK1/2 hyperphosphorylation in embryonic hearts has been shown previously to result in congenital cardiac defects resulting from altered cardiomyocyte cell cycling [58] .

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