Strikingly, the metabolomic study revealed a highly significant correlation between nonfasting glucose and β-hydroxypyruvate levels for mice regardless of diet or genotype, indicating individual intra- and intergroup variants and outliers exhibit an appropriate correlation. β-Hydroxypyruvate induces apoptosis of astroglial but not liver or kidney cells ( 41 ).
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Global biochemical profiling identifies β-hydroxypyruvate as a potential mediator of type 2 diabetes in mice and humans.
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