Other proinflammatory mediators, such as CXCL10, IL-10, MMP2 and TNF, also showed a decreasing trend in hOXR1-MSCs treated mice compared with the control mice (data not shown).
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Delivering Oxidation Resistance-1 (OXR1) to Mouse Kidney by Genetic Modified Mesenchymal Stem Cells Exhibited Enhanced Protection against Nephrotoxic Serum Induced Renal Injury and Lupus Nephritis.
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