Homocysteine thiolactone and N-homocysteinylated protein induce pro-atherogenic changes in gene expression in human vascular endothelial cells.

Top five highly significant [−log( P value) = 20–31] pathway categories affected only by Hcy-thiolactone were ‘chromatin organization,’ ‘one-carbon metabolism,’ ‘lipid localization,’ ‘lipoprotein metabolic processes,’ and ‘lipid and fatty acid transport’ (Fig. 3 a).

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