GTSE1 expression represses apoptotic signaling and confers cisplatin resistance in gastric cancer cells.

Although cisplatin treatment induced an inhibitory effect in the colony formation ability of both AZ521-con and AZ521-kd cells, the decrease of colony formation in GTSE1 knocked-down cells appeared to be more drastic and highly significant ( p = 0.002) as compared to the control cells (Figure 4b ).

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