While setting up the model, we discovered that the hyperplasia of glomerular mesangial cells and cellular matrix and the levels of clinical biochemical indicators, such as 24-h urine proteins and serum creatinine, displayed an increasing trend, suggesting that the model was successfully set up.
← all excerpts
Protective Effect of Triptolide against Glomerular Mesangial Cell Proliferation and Glomerular Fibrosis in Rats Involves the TGF- β 1/Smad Signaling Pathway.
1
—
—